hypoglycemia and dysinsulinism are three of the most common metabolic
dysfunctions afflicting Americans today. It has been estimated that there are
well over three million diagnosed diabetics and at least three times as many
more that remain undiagnosed. At least 50 percent of our population suffer from
the symptoms of hypoglycemia to some extent.
is now a leading cause of blindness and kidney disease. Hypoglycemia is
recognized as an important cause of fatigue, alcoholism, crime, hyperkinetic
behavior, learning disabilities, behavioral disorders, anxiety, depression,
allergies and many other physical, mental and emotional problems.
concepts allow for a much deeper and correction of these common conditions.
MECHANISM OF BLOOD SUGAR UTILIZATION
discussing the various dysfunctions associated with deviations in blood sugar
metabolism, it will be helpful to review the mechanism of glucose regulation in
AND RELEASE OF GLUCOSE
sugars and carbohydrates are absorbed as simple sugars and pass to the liver,
where some sugar is allowed to enter the general circulation. However, the
majority of ingested glucose is normally converted to glycogen, the storage form
of sugar and is stored mainly in the liver.
reduction in the circulating blood sugar levels normally stimulates
glucocorticoid hormone production by the adrenal glands. One effect of increased
cortisone output is to cause the liver to reconvert glycogen into glucose and
release it into the blood. In this way, optimum blood sugar levels are normally
of the adrenal glands, either a deficiency of adequate hormone production or an
excess, seriously interferes with normal blood sugar regulation.
adrenal function requires the presence of certain nutrients such as manganese,
zinc, vitamin C, pantothenic acid and vitamin E. In addition, the
adrenal hormones are synthesized from cholesterol, so that a deficiency in the
synthesis of cholesterol will impair adrenal function.
gland activity is also a function of the metabolic or oxidation rate, which
is in turn, is governed by the nutritional balance and by the activity of the
sympathetic nervous system.
is a polypeptide hormone, which is necessary for the utilization of glucose in
the body. The function of insulin allows glucose to pass through the cell
presence of excess sugar in the blood stimulates the release of insulin from the
beta cells in the Islets of Langerhans of the pancreas. Zinc is
closely involved with insulin synthesis, secretion and function.
has a complicated relationship to insulin. Research reveals the following:
At the pH of the pancreas, insulin can only be crystallized in the presence of
zinc, cadmium, cobalt and nickel ions.
Crystalline insulin is coated with zinc, the more zinc that can be made to
adhere to the insulin molecule, the longer the duration of insulins action.
There is evidence that zinc is utilized in the beta cells of the pancreas to
both store and release insulin as required. Release of insulin from the
beta cells is accompanied by a loss of zinc.
Zinc seems to have a similar action to insulin, in stimulating uptake of glucose
by adipose tissue. A deficiency of zinc results in reduced uptake of glucose by
Injection of Dithiazane, a zinc chelating agent, produces diabetes in
Pancreatic tissue of diabetic individuals has been shown to have one-third the
zinc concentration of controls.
Zinc may be necessary for retention of chromium, which plays a vital role in
release of insulin is facilitated by calcium and inhibited by magnesium.
Thus the proper ratio of calcium to magnesium is critical for optimal
acts synergistically with insulin.
deficiency is widespread in the United States and Canada, due to excessive sugar
and carbohydrate intake and to soil depletion. Chromium definitely acts as a
synergist with insulin. Research by Mertz (1969), indicates at least five
Chromium could stabilize the structure of the insulin molecule in its most
Chromium could increase the binding of insulin to the tissues.
Chromium could be a co-factor for a cell membrane transfer mechanism of insulin.
Chromium could facilitate the initial reaction between insulin and a specific
cell membrane receptor site.
is utilized in the form of Glucose Tolerance Factor, in which chromium is
combined with four amino acids. A deficiency of chromium definitely results in
impaired insulin function, which may result in either a hypoglycemia syndrome or
OF NUTRIENTS ACROSS CELL MEMBRANES
transport of nutrients across the cell membrane is regulated, in part, by the sodium
pump mechanism, which regulates the electrical potential and osmotic pressures
across cell membranes. The proper amount and balance between sodium and
potassium in the cell environment are therefore, also critical to the
utilization of glucose.
cell membrane permeability is also regulated by calcium. Calcium
acts as a stabilizer of cell membrane potential. Excess calcium, for example,
renders the cells less permeable to glucose as well as to insulin.
OF GLUCOSE WITHIN THE CELLS
inside the cells, the combustion or utilization of glucose depends on the
availability of enzyme co-factors and activators. Among the most important of
these is magnesium, which is a vital component of numerous intra-cellular
enzymes and manganese, vitamins B1, B2, B3 and B6, which are
required for the glycolysis cycle, the first part of the energy-producing
copper, magnesium, B1 and B2 are
required as co-factors and activators of the Krebs cycle and the electron
transport system, which completes the burning of glucose, to produce energy in
the form of adenosine-triphosphate (AlP).
due to a deficiency of any of the above nutrients, at any step of this process,
will result in symptoms of diabetes or hypoglycemia, depending on at exactly
what step in the energy cycle that the blockage occurs.
hormone also plays a vital role, in
as much as thyroxin regulates the rate of combustion of glucose within the
OF CAUSES OF BLOOD SUGAR DISTURBANCES
possible causes of diabetes and hypoglycemia can include any combination of the
A deficiency of manganese, zinc, B-complex, vitamin C, or vitamin E; impaired
adrenal function, affecting cortisone secretion, which in turn, affects glycogen
release from the liver.
lmbalanced oxidation rate affects glucocorticoid hormone release.
Zinc deficiency prevents adequate production of insulin.
A low calcium/magnesium ratio diminishes the release of insulin from the beta
cells of the pancreas.
A deficiency of chromium prevents proper transport and attachment of insulin to
cell receptor sites.
Imbalances in sodium, potassium, calcium and magnesium interfere with cell
membrane function, preventing glucose and/or insulin from entering the cells.
The body may then compensate by raising the sugar level to force sugar into the
A deficiency of enzyme co-factors in the Krebs and glycolysis cycles impairs
glucose utilization. The body again may compensate for low energy production by
raising blood sugar levels in an attempt to force more sugar into the cells.
Abnormal thyroid activity affects the rate of sugar combustion within the cells.
With this overview in mind, we will focus on each metabolic dysfunction
condition in greater detail.
simply stated, is defined as low blood sugar. However, many individuals with
normal or even high glucose tolerance tests frequently manifest many of the
symptoms of low blood sugar.
occurs because there is confusion about the definition and meaning of
hypoglycemia. While the word hypoglycemia strictly refers to low
glucose in the blood, many individuals use the term loosely to
designate a specific group of symptoms arising from inadequate glucose
metabolism. Inadequate glucose metabolism can have many causes, one of which is
a decreased availability of glucose in the blood. Other causes include: 1) low
levels of glucose in the cells (cellular hypoglycemia) and 2) adequate levels of
glucose in the cells, but inadequate combustion of that glucose.
help dispel the confusion, in this article hypoglycemia refers to the syndrome
of inadequate glucose utilization by the cells, for any reason. The reason for
defining hypoglycemia as a cellular problem, is that it is within the cells that
glucose is utilized.
this definition, it becomes obvious that the use of a blood test for
hypoglycemia is not always reliable. Many individuals who have had a 5-hour
glucose tolerance test are well aware of this inadequacy. Their test report was
normal, but they suffered all the symptoms of low sugar during the test.
OF THE HYPOGLYCEMIA SYNDROME
the blood serum test is not reliable, what indicators can be used? Several
simple questions are often a reflective indicator:
Ask yourself how you feels if you goes without eating more than about 4 hours.
The hypoglycaemic usually cannot do this without experiencing symptoms of
fatigue, extreme hunger, weakness, irritability, or mental confusion.
Ask yourself how you feel after you ingest sugar or sweets, without balancing it
with other types of foods such as fats or proteins. In some forms of
hypoglycemia, eating sugar or simple carbohydrates will bring on distressing
symptoms. Another screening tool often used as an indicator is tissue mineral
studying thousands of cases of hypoglycemia, Dr. Paul Eck discovered certain
mineral patterns associated with this condition. These are referred to as trends,
because they indicate a tendency, but are not diagnostic in and of
themselves. These trends can only be read out on a hair analysis performed
without washing the hair and with proper laboratory controls. Trends include:
Imbalance in the calcium/magnesium ratio, especially a ratio between 3.6/I and 5.3/I,
or 8.0/1 and 9.7/1.
Slow oxidation, especially with very low levels of potassium (below 5.0 mgs/%),
or a very high calcium/potassium ratio (higher than 100/1 mgs./%).
Fast oxidation, especially if it is extreme, and/or accompanied by a low zinc
level and imbalanced (high or low) calcium/magnesium ratio or a high
following section explains the rationale for these empirically observed trends.
Oxidation and Excessive Gluconeogenesis
oxidation refers to a mineral pattern of low tissue calcium and magnesium levels
relative to sodium and potassium levels.
oxidation, with a high sodium/potassium ratio, is associated with the alarm stage
of stress, characterized by rapid metabolism of food and over-activity
of the thyroid and adrenal glands. True fast oxidation is found in approximately
10% of the population and is associated with one specific type of hypoglycemia.
increased adrenal gland activity associated with fast oxidation causes a state
of continuous gluconeogenesis, or conversion of glycogen to glucose. The
end-result is a mild to severe depletion of liver glycogen reserves.
of the thyroid gland causes excessively fast burning of glucose, which results
in rapid depletion of liver glucose reserves. When additional extra glucose is
suddenly required (usually due to stress) and the glycogen reserves of the liver
are depleted, the result is the manifestation of acute symptoms of low blood
fast oxidizer is prone to what is referred to as reactive hypoglycemia,
in which blood sugar levels, after a meal, rise dramatically and then fall
precipitously. This type of hypoglycemia can readily be brought on by certain
foods or activities that speed up the oxidation rate. Sweets, alcohol,
coffee, overwork, exercise, or stress of any kind can readily trigger acute
symptoms of hypoglycemia in a fast oxidizer. Various food supplements such as
vitamin C, vitamin E and B-complex may, because of their stimulatory affects on
metabolism, produce the same effect.
in the Slow Oxidizer;
Gluconeogenesis and Impaired Membrane Transport
oxidation is a condition indicated on a tissue mineral analysis readout by high
calcium and magnesium levels, relative to sodium and potassium levels. Slow
oxidation represents a state in which the thyroid and adrenal glands are
relatively under-active. Adrenal insufficiency results in the production of less
than normal amounts of giucocorticoid hormones.
with a need for an increase in blood sugar, the body cannot secrete adequate
levels of cortisone to stimulate sufficient conversion of glucose from glycogen
in the liver; the end result is hypoglycemia.
slow oxidizer has an additional problem that contributes to the hypoglycemia
syndrome; transport of glucose across the cell membranes is impaired, due to low
levels of sodium and potassium and an elevated calcium level. Therefore, even if
glucose levels in the blood are adequate, glucose may not be transported in
adequate amounts across the cell membranes.
slow oxidizers are the group of individuals who may exhibit many of the symptoms
of hypoglycemia, while reporting normal, or near-normal, glucose levels on a
5-hour glucose tolerance test.
slow oxidizer has a third problem that frequently may result in symptoms
associated with the hypoglycemia syndrome. In slow oxidation, the burning of
glucose within the cells is more or less dysfunctional and inefficient. Glucose
may enter the cells, but is not metabolized adequately, due to a deficiency of
enzyme co-factors and/or diminished thyroid hormone activity. These factors can
initiate hypoglycemia symptoms, even if glucose is available in the blood and is
adequately transported into the cells.
the slow oxidizer, hypoglycemia symptoms are often chronic. Symptoms include;
chronic fatigue, a constant craving for sweets, mental confusion and depression.
Symptoms are frequently aggravated or triggered by fatigue, which further
reduces adrenal activity. Eating certain foods, which slow the oxidation rate
such as; dairy foods, high fat diet, or a low protein diet may also trigger
use of calcium, magnesium and zinc supplements, vitamin A, vitamin C and copper
supplements may also aggravate hypoglycemic symptoms inasmuch as these nutrients
tend to lower the metabolic rate.
temporarily alleviate hypoglycemic symptoms by temporarily stimulating adrenal
gland activity. However, in the long run, unless adrenal activity is
sufficiently restored, vigorous exercise can worsen hypoglycemia associated with
slow oxidation by causing increased exhaustion of the adrenal glands.
mellitus literally refers to sugar in the urine. As with hypoglycemia, this term
tells us nothing about the cause of this increasingly common
metabolic dysfunction. In more modern terms, diabetes refers to a specific
pattern of glucose tolerance, in which the blood sugar level rises excessively
and remains elevated for several hours, above the normal glucose tolerance
is often viewed as a simple deficiency of insulin. Nothing could be further from
the truth. The concept that diabetes is due to a deficiency of insulin is too
simplistic and outdated. Diabetes occurs with normal or even elevated insulin
levels and is due to a variety of causes and mechanisms.
in the Fast Oxidizer
fast oxidizer burns glucose too rapidly and therefore has an increased demand
for insulin. The fast oxidizer is also particularly prone to a zinc deficiency,
because zinc is lost as the body goes into the alarm stage of
stress, which characterizes fast oxidation.
high insulin requirement and a tendency to a zinc deficiency can combine in the
fast oxidizer to produce a deficient insulin type of diabetes. The deficiency of
zinc is intimately associated with insufficient production or diminished release
low calcium level also characterizes fast oxidizers. Calcium, in optimal
amounts, is required for the release of insulin and thus another type of insulin
deficiency diabetes results from a calcium deficiency.
type of diabetes is further accentuated when magnesium levels are high relative
to calcium levels, inasmuch as magnesium inhibits insulin release.
in the Slow Oxidizer
oxidizers may also suffer from a zinc deficiency, or low calcium to magnesium
ratio, with the same consequences as the fast oxidizer. However, other
dysfunctions prevail in the slow oxidizer, which predisposes one to diabetes.
oxidation is characterized by impaired or blocked transport of glucose across
cell membranes and inadequate utilization of glucose within the cells. An
elevated tissue calcium and magnesium level together with a deficiency of sodium
and potassium can result in an impairment of cell membrane permeability. In
these cases, insulin levels are normal or even high; the body may raise insulin
levels to help force glucose into the cells.
passing into the cells fails to be metabolized properly in the glycolysis cycle
in slow oxidation. Failure of sufficient glucose to be transported across the
cell membrane may be due to a deficiency of manganese, bio-unavailable
magnesium, toxic metals, deficiency of B vitamins or decreased thyroid hormone
activity. The body may attempt to compensate for a reduction of energy
production, again by raising blood sugar and insulin levels.
oxidizers are usually deficient in manganese and frequently deficient in
chromium and zinc as well. While the zinc level may appear to be normal, zinc
is low relative to copper if the zinc/copper ratio is less than 5/1.
calcium/magnesium and sodium/potassium ratio imbalances can also occur in the
slow as well as the fast oxidizer.
AND HYPOGLYCEMIA SYNDROME CAN COEXIST
common misconception is that a person is either hypoglycaemic or diabetic. The
fact is, one of the major symptoms of diabetes is extreme fatigue and diabetics
generally suffer from cellular hypoglycemia. The reasons for this
Since insulin is necessary for glucose absorption into the cells, a deficiency
of insulin blocks adequate glucose from entering the cells, resulting in a
cellular hypoglycemia syndrome.
Some cases of diabetes result from or are aggravated by a chromium deficiency,
which impairs attachment of insulin to cell membrane receptor sites. The end
result is an impairment of glucose transport into the cells, resulting in low
cellular glucose levels.
Some cases of diabetes are due to an excess bio-unavailability of tissue
calcium, which impairs the transport of glucose across the cell membranes. The
body raises blood glucose levels in these cases, to help force glucose into the
cells. In these cases also, there can be a cellular hypoglycemia, in spite of
excess glucose in the blood.
Some cases of diabetes are associated with, or are aggravated by. a manganese
deficiency. In these cases, adequate levels of insulin may be released, but not
transported in sufficient amounts to receptor sites on the cell membrane. Again,
this causes inadequate glucose absorption into cells and cellular hypoglycemia
A deficiency of manganese also inhibits the activity of the enzyme pyruvate
carboxylase, which is necessary for sugar and carbohydrate metabolism in the
glycolysis cycle. Even if adequate glucose reaches the cells, energy production
from the glucose is inadequate, leading to symptoms of hypoglycemia.
Slow-oxidizer diabetes is associated with inadequate thyroid function. A
deficiency of adequate cellular thyroid hormones causes slow burning of glucose,
even if glucose is readily available.
recognition of diabetes is vitally important because diabetes is a primary cause
of so many other metabolic dysfunctions, including kidney disease,
atherosclerosis, blindness and diabetic neuropathy.
simple finger stick blood test, or a single urine evaluation is clearly
inadequate, because sugar levels fluctuate greatly with diet and physical
5-hour glucose tolerance test is the standard diagnostic test for diabetes.
However by the time this test is performed, a person may have had diabetes for
several years and damage of vital organs and numerous other metabolic
dysfunctions may have begun.
problem can be largely avoided if early signs and symptoms of diabetes are
SIGNS AND SYMPTOMS OF DIABETES
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Extreme exhaustion not related to activity.
As glucose levels increase, energy levels
often fall off drastically. Periods of utter exhaustion occur from out of the
blue. Fatigue can occur suddenly, without any warning.
Loss of Libido - Related to glucose related zinc deficiency.
Frequent urination - Glucose acts as an osmotic diuretic, taking
water with it as it is eliminated in the urine.
Excessive thirst, due to a loss of water from excessive urination.
Burning sensation on urination.
Thickened toenails, with fibrous material underneath.
Discoloration of the toenails. Nails first turn yellow, then brownish
as the condition worsens.
Increased curvature of the nails and tendency for ingrown toenails.
Toenails may actually fall off from the base in some cases.
Encrustation of the eyelashes. At times,
the eyes are glued shut in the morning and pus may exude from the eyes,
particularly upon awakening.
TRENDS DERIVED FROM A HAIR ANALYSIS
of diabetes from a hair analysis represent another valuable way of early
detection of diabetes. The most important mineral trends are:
A low sodium to potassium ratio (less than 1.5/1). This ratio is
intimately associated with excessive protein catabolism and glucocorticoid
secretion. In diabetics, glucose cannot be oxidized in sufficient amounts to be
converted to energy-rich adenosine-triphosPhate (ATP). As a result, the body is
forced to convert protein to glucose to maintain energy production.
A high or low calcium to magnesium ratio (greater than 10/1 or less than 3.3/1),
especially when combined with a low sodium to potassium ratio. Since calcium
and magnesium are involved in insulin release, an imbalance in this mineral
ratio is indicative of blood sugar dysfunction.
Low chromium, zinc and/or manganese levels, especially when combined with
the previously listed trends. As explained earlier, each of these minerals is
intimately involved in insulin function and carbohydrate metabolism.
iron (above 5.0 mgs/l), or copper (above 3.5 mgs/l) levels, especially when
combined with a low sodium to potassium ratio. An elevated iron level is an
excellent indicator of excessive tissue protein catabolism. Iron is being
released due to excessive breakdown of tissue cells. Iron, by being also
antagonistic to chromium, can cause a chromium deficiency. Excess copper
antagonizes zinc and is associated with protein catabolism as well.
mineral analysis can be utilized as an excellent screening tool. If a person has
one or more of the above trends and exhibits some of the physical symptoms
listed above, it would be prudent to check urine or blood sugar levels.The more
mineral trends and/or symptoms, the more likely a blood sugar intolerance is
present. For instance, a low chromium level, together or coupled with a
sodium/potassium inversion, indicates diabetes more than either of these
particular advantage of tissue mineral analysis methodology is that you can
begin correction of the mineral imbalances, even before the glucose tolerance
test reveals a problem.
ROLE OF TOXIC METALS IN HYPOGLYCEMIA AND DIABETES
metals can directly or indirectly play a role in the causation of both diabetes
and hypoglycemia. A toxic metal may precipitate blood sugar dyscrasia* by:
Interfering with absorption of essential minerals and creating deficiencies of
Occupying a binding site on a metalloenzyme, preventing the preferable mineral
from attaching to that site and thus inhibiting vital enzymatic function.
Altering the oxidation rate resulting in either fast or slow oxidation.
Forcing another type of compensation or adaptation to the stress generated by
the toxic metal, resulting in an alteration of a critical ratio, such as the
calcium/magnesium or sodium/potassium ratio. These mineral ratio imbalances then
cause a blood sugar dysfunction.
* an abnormal or
physiologically unbalanced state of the body.
TOXIC METALS ASSOCIATED WITH HYPOGLYCEMIA AND DIABETES
specific diabetic pattern is associated with elevated tissue iron levels. The
most probable mechanism in high-iron diabetes is that iron may cause a chromium
deficiency thereby hindering the utilization of insulin. Iron is also
antagonistic with manganese and it is possible that this is a factor as well.
Cadmium antagonizes zinc and can replace zinc in
vital metallo-enzyme binding sites.
antagonizes zinc, raises calcium and slows oxidation by impairing adrenal and
thyroid function. Many slow oxidizers with hypoglycemic symptoms can be traced
to an elevated copper level.
have mentioned that excess tissue calcium levels interfere with cell membrane
permeability and may be responsible for inadequate transport of glucose into
may act indirectly to produce symptoms of hypoglycemia, because mercury
poisoning interferes with normal copper metabolism.
The term dysinsulinism refers to a glucose
tolerance curve that exhibits characteristics of both hypoglycemia and diabetes.
Dysinsulinism is considered a transition stage between the two, in which there
may be alternating symptoms of both hypoglycemia and diabetes. According to Dr.
Ecks research, dysinsulinism is associated with a calcium/magnesium ratio
between 3.3/1 and 3.6/1 or between 9.8/1 and 10.0/1.
OF HYPOGLYCEMIA IN THE FAST OXIDIZER
requires slowing the oxidation rate:
excessive corticosteroid output accomplishes two purposes:
Reduces gluconeogenesis. thus restoring glycogen reserves in the liver, which
can be called upon to bolster blood sugar levels.
Restores a reserve of corticosteroid hormones. The combination of more available
cortisone and more available glycogen allows adrenal cortisone output to be
increased upon demand and ensures a reserve of glycogen to be drawn upon when
excessive thyroid activity helps slow the excessive rate of glucose
metabolism in the fast oxidizer, thereby reducing the possibility of depletion
of glucose reserves.
low calcium levels and reducing excessively high potassium and sodium levels,
slows transport of glucose and thyroid hormone into the cells, which also
contributes to slowing the combustion of glucose.
diet high in fat, particularly animal fats, tends to slow the excessive
oxidation rate. There may be several mechanisms for this:
Fats are digested and absorbed slowly from the intestine. Therefore, fats do not
exert a stimulatory effect on metabolism the way rapidly absorbed sugars do.
Fat consumption also enhances absorption of the fat-soluble vitamins A and
D. Vitamin A acts synergistically with zinc to lower
sodium levels, slowing the oxidation rate and vitamin D increases calcium
absorption from the gut, also tending to slow the oxidation rate, by reducing
excessive glucocorticoid activity.
Fast oxidizers tend to have difficulty with that part of the energy production
cycle known as the Krebs cycle. Fats provide a high level of acetates, which
play an essential role in this part of the energy production system.
Fat is also a high-energy food as compared to either carbohydrates and proteins,
yielding 9 calories per gram. Fast oxidizers benefit from this higher energy
Carbohydrates and Sugars
Carbohydrate foods frequently contain phytates, which lower calcium, magnesium
and zinc levels. Calcium, magnesium and zinc are required to slow an excessive
oxidation rate, so a low carbohydrate diet tends to spare these essential
Sugars and carbohydrates are more rapidly absorbed and have a more stimulatory
effect on metabolism.
Nutrients such as calcium, zinc, magnesium, copper, vitamin A, D, B2, B12,
choline and inositol are also helpful to slow an excessive oxidation rate.
OF HYPOGLYCEMIA IN THE SLOW OXIDIZER
solution for these individuals is to reactivate adrenal and thyroid activity.
Increased secretion of adrenal cortical hormones allows release and conversion
of more glycogen to glucose from liver storage sites, thus improving blood sugar
Increased adrenal function raises sodium and potassium levels, which improves
transport of glucose into cells.
Increase in thyroid hormones promotes more efficient combustion of glucose
within the cells.
Improved adrenal hormone reserves permit increased secretion of cortisone, when
glucose demands increase suddenly as occurs during stress, or during
diet high in the low-fat proteins, adequate in complex carbohydrates and low in
fat tends to speedup the rate of metabolism. Carbohydrate is relatively rapidly
absorbed and phytates as contained in grains favor faster oxidation. Protein in
the diet stimulates glandular function, raises sodium and lowers magnesium, thus
favoring fast oxidation.
fat slows the rate of metabolism and is therefore ideally kept to a minimum.
A, vitamin C, vitamin E, the B-complex (particularly vitamin B-1) and manganese
specifically enhance adrenal function. Manganese, the B-complex and vitamin E
enhance thyroid and intracellular glucose metabolism.
is particularly essential for protein metabolism and is frequently deficient in
the slow oxidizer, in part due to an elevated copper level.
is required and recommended to help lower excessively high calcium levels, thus
improving thyroid function and transport of nutrients across the cell membranes.
toxic metals are present such as cadmium, copper, mercury and nickel. These
toxic metals block various enzyme functions and must be slowly removed by giving
appropriate mineral antagonists, oral chelating agents and by balancing the
OF DIABETES IN THE FAST OXIDIZER
requires improving insulin secretion, transport and utilization.
low calcium, zinc, chromium and manganese levels serve to improve insulin
synthesis, secretion and transport.
the oxidation rate reduces excessive adrenal cortical hormone production, which
reduces conversion of glycogen to glucose and contributes to lowering of blood
same general dietary recommendations apply for the fast oxidizer diabetic as for
the fast-oxidizer hypoglycemic.
has a sparing action on insulin, because fat slows the emptying time of the
stomach, causing slower absorption of sugars into the blood.
the quantity of starches and sugars in the diet reduces the influx of sugars
into the blood from the intestine and hence reduces insulin requirements.
type diabetics benefit from calcium, both because it slows the rate of
oxidation, thus sparing insulin and because calcium promotes the release of
insulin from the beta-islet cells of the pancreas.
zinc and chromium are added to all diabetics programs, two tablets of each
twice a day, because of the importance of these minerals in insulin production
diabetes in the fast oxidizer is associated with elevated iron levels and/or
elevated cadmium levels. If this is the case, specific nutrients may be given as
indicated to reduce elevated iron and cadmium levels. Copper, manganese and
chromium, in excess, interfere with iron absorption. Calcium, selenium, vitamin
C, copper and zinc may be given as indicated to reduce excessive cadmium levels.
OF DIABETES IN THE SLOW OXIDIZER
slow-oxidizer diabetic should follow the same general dietary guidelines as have
been described for the slow-oxidizer hypoglycemic. Since the cause of
slow-oxidizer diabetes has more to do with inadequate transport of glucose into
the cells and inadequate insulin utilization, rather than inadequate production
of insulin, different measures are indicated.
of simple sugars is less critical in the slow-oxidizer diabetic than in the fast
oxidizer. However, refined sugars should definitely be avoided as they lack the
essential mineral elements.
should be avoided inasmuch as they slowdown the metabolic rate further.
proteins tend to improve the metabolic rate by stimulating adrenal and liver
function. Phytates contained in grains assist in lowering excessively high
and sodium-raisers (nutrients which serve to increase sodium levels) are
recommended to reduce elevated tissue calcium levels because high tissue calcium
levels interfere with both insulin secretion and glucose transport across cell
membranes. Sodium and potassium levels must also be increased to improve
functioning of the sodium-potassium pump mechanism.
information on foods and nutrients that increase potassium levels click here
above measures help glucose to enter body cells. If glucose can be made
available to the cells, the compensation of an elevated blood sugar will no
longer be necessary.
is a critical element for the slow oxidizer individual; not only because it is
involved in insulin transport, but because it raises ones sodium level. This
in turn, enhances the transport of glucose into the cells and improves both
adrenal and thyroid activity. Thyroid hormone speeds up the rate of burning of
glucose within the cells.
if transport across the cell membrane is adequate, glucose may build up within
the cells, if it is not adequately metabolized.
vitamins and vitamin E are also required to enhance thyroid activity.
and zinc are also recommended for the slow-oxidizer diabetic in the dose of one
tablet of each, twice a day. An elevated copper level in the slow oxidizer often
interferes with zinc metabolism. Chromium levels tend to be borderline-low in
all oxidation types.
and thyroid protomorphogens support glandular function and assist in increasing
a slow oxidation rate.
OF HYPOGLYCEMIA AND DIABETES
same principles of nutrient deficiencies, toxic metals and biochemical
imbalances, can be applied to a person to prevent as well as to treat
hypoglycemia and diabetes. Prevention is really the wiser choice. It is far less
costly and less time-consuming than treating a full-blown case of illness.
eating a high-quality diet that contains adequate manganese, zinc, chromium,
copper and other essential nutrients are one simple way of prevention. However,
today even our natural foods are deficient in these vital elements, due to
agricultural methods, storage and processing methods. Good diet alone is not
sufficient assurance against the development of hypoglycemia and
analysis provides a simple, relatively inexpensive, early screening test in
which the trends for illness can be recognized; often several years before
symptoms become manifest. Also, the test provides us with direction for action
to correct the nutrient imbalances that are currently present, to avoid more
The Time Factor
correction process for both hypoglycemia and diabetes depends on many factors.
Requiring in rare instances as little as several months, but more likely
requiring up to several years, depending upon which nutrients are deficient and
how severe the mineral ratio imbalances. There is, at present, no way to tell
how long will be required from a single hair analysis, or from symptoms alone.
and Passwater in their book, Nutrition. Trace Elements. and Hair Analysis,
state that it requires 2 years to restore to normal, low chromium levels. In
some individuals, it is also necessary to restore manganese, zinc and numerous
other nutrients, as well as chromium.
elimination of toxic metals must also be slow and unforced. Rapid removal of
toxic metals tends to upset body chemistry, because the toxic metals frequently
serve an adaptive and supportive function. The body will seldom allow their
releases until vital mineral reserves have been built-up sufficiently and the
body chemistry is stable enough to withstand the release of toxic crutches.
the correction process, a diabetic may pass through a stage of hypoglycemia.
occurs because hypoglycemia is an earlier stage of metabolic dysfunction and as
the mineral patterns return toward normal, the body may temporarily pass back
through a phase of hypoglycemic mineral ratios and levels. This ordinarily is no
cause for alarm and regular retesting will minimize any possible symptoms.
is also possible to pass through another diabetic pattern during the correction
process. This also is temporary and due to the complex changes in the mineral
balance that occur as toxic metals are removed and essential minerals act to
compensate and adapt to the changes taking place in the body chemistry.
levels will also vary during the recovery process. Since both diabetic and
hypoglycemic individuals suffer from a periodic energy loss, they may become
dismayed if, after a period of improvement, an energy loss later occurs. Again,
this is no cause for alarm. Energy loss may occur on the program at any point,
Use of energy within the body for restoring vital body functions, causing
diminished availability of energy for external activity.
Rebalancing of the body chemistry, causing a temporary slowdown in metabolism.
Occasionally, metabolism may speed up greatly, also resulting in a temporary
Change in another mineral level or ratio which is intimately related to energy
production, such as a change in iron, copper, manganese, sodium, or calcium
hypoglycemia syndrome and diabetes are complex biochemical dysfunctions, whose
causes are multiple in nature. We have attempted to present the underlying
pathophysiology of these metabolic dysfunctions, as well as to explain the
principles of a new nutritional approach to their correction.
the use of tissue mineral analysis and application of the principles of the mineral
balancing approach to correction of body chemistry; we have had excellent
success in both preventing and reversing many of the signs and symptoms of these
C 1987 - The Eck Institute of Applied
Nutrition and Bioenergetics Ltd.